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İçerik, diyabetin türleri (tip 1, tip 2, LADA, monojenik diyabetler), tanı algoritması, oral antidiyabetik ilaçlar, insülin kullanımı, komplikasyonlar (mikrovasküler, makrovasküler, nöropati, hipertansiyon, dislipidemi) ve tedavi stratejileri gibi konuları sistematik bir şekilde ele almaktadır.","Video özellikle tıp öğrencileri, klinik pratikte çalışan doktorlar, dahiliyeciler ve acil servis çalışanları için hazırlanmıştır. İçerikte her ilaç grubunun etki mekanizmaları, yan etkileri, kontra-endikasyonları ve farklı sağlık durumlarındaki etkileri detaylı olarak açıklanmakta, ayrıca diyabet hastalarının düzenli takip edilmesi gerektiği vurgulanmaktadır."]},"endTime":7662,"title":"Türk Endokrinoloji ve Metabolizma Derneği'nin Yeni Diyabet Kılavuzu Eğitim Videosu","beginTime":0}],"fullResult":[{"index":0,"title":"Giriş ve Kılavuz Hakkında Genel Bilgiler","list":{"type":"unordered","items":["Dr. Atilla Uslu, Türk Endokrinoloji ve Metabolizma Derneği'nin Haziran 2024 tarihinde yayınladığı yeni diyabet kılavuzunu tartışmak için burada.","Diyabetik hastalar her uzmanlık alanında ve klinik pratikte bulunur, bu nedenle bu hastalara optimal bakımı vermek ve gerektiğinde konsültasyon yapmak zorunludur.","Kılavuz 320 sayfalık geniş kapsamlı bir belgedir ve tıpta uzmanlık sınavı, yan dal uzmanlık sınavı ve klinik pratiğe yönelik bilgilere odaklanacaktır."]},"beginTime":0,"endTime":83,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=0&ask_summarization=1"},{"index":1,"title":"Video Hedef Kitlesi ve Kullanım Bilgileri","list":{"type":"unordered","items":["Video, 1. ve 2. sınıf tıp öğrencileri için değil, tıp öğrencileri ve sahada çalışan doktorlar için hazırlanmıştır.","Kullanılan tüm slaytlar kılavuzdaki tablolardan ve şemalardan alınmıştır, ayrıca kendi doktor dahiliye konu kitabından da bazı bilgiler kullanılmıştır.","Slaytlar Telegram kanalından ulaşılabilir ve video chapterlara ayrılarak izlenebilir."]},"beginTime":83,"endTime":171,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=83&ask_summarization=1"},{"index":2,"title":"Diyabet Tanı Kriterleri","list":{"type":"unordered","items":["Diyabet tanı kriterleri: açlık plazma glikozu 126 mg/dL veya üzerinde, rastgele plazma glikozu 200 mg/dL ve üzerinde, klasik diyabet semptomları eşlik ediyorsa veya hemoglobin A1C %6,5 veya üzerinde ise.","OGTT (Oral Glikoz Tolerans Testi) özellikle gebelerde popülerdir; 75 gram glikoz verildiğinde ikinci saat plazma glikozu 200 mg/dL ve üzerinde ise diyabet tanısı konulur.","Bazı durumlarda ne diyabet ne de normal diyabete sahip olunabilir, bu durumda prediyabet olarak isimlendirilir ve bozulmuş açlık glikozu (100-125 mg/dL), bozulmuş glikoz toleransı (140-199 mg/dL) ve yüksek risk grubu (HbA1C %5,7-6,4) olarak alt gruplara ayrılır."]},"beginTime":171,"endTime":330,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=171&ask_summarization=1"},{"index":3,"title":"Hemoglobin A1C ve Diyabet Semptomları","list":{"type":"unordered","items":["Hemoglobin A1C, glikozillenmiş hemoglobin olarak adlandırılır ve hemoglobinopati, kanama, hemodiyaliz veya eritropolitin tedavisi verilen hastalarda yanlış sonuçlar verebilir.","Tip 1 diyabetik hastalarda açlık plazma glikozu daha iyi bir tanı kriteridir çünkü tip 1 diyabet genellikle akut olarak ortaya çıkar ve hiperglisemi birkaç aylık değildir.","Klasik diyabet semptomları poliüri (çok idrar), polidipsi (çok su içme) ve polifaji (çok yemek) olup, ayrıca ağız kuruluğu, gece idrara çıkma, halsizlik ve çabuk yorulma gibi semptomlar da görülebilir."]},"beginTime":330,"endTime":468,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=330&ask_summarization=1"},{"index":4,"title":"Gestasyonel Diyabet","list":{"type":"unordered","items":["Gestasyonel diyabet, gebelerde görülen bir diyabet tipidir ve tedavi edilmezse hem anne hem de bebek için ciddi komplikasyonlar yaratabilir.","Gestasyonel diyabet hastalarının daha sonraki hayatlarında kalıcı diyabet olma riski normal popülasyondan daha yüksektir.","Gestasyonel diyabet taraması 24-28. haftalar arasında yapılır; iki aşamalı testte ilk aşamada 50 gram glikoz verilir ve birinci saatte 140 mg/dL ve üzerindeyse ikinci basamağa geçilir, tek aşamalı testte ise 75 gram glikoz verilir ve ikinci saatte 153 mg/dL ve üzerindeyse veya birinci saatte 180 mg/dL ve üzerindeyse gestasyonel diyabet tanısı konulur."]},"beginTime":468,"endTime":533,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=468&ask_summarization=1"},{"index":5,"title":"Diyabetin Tiplendirmesi","list":{"type":"unordered","items":["Diyabet iki ana tip olarak tip bir diyabet ve tip iki diyabet olarak bilinir, ancak Dünya Sağlık Örgütü'nün 2009 yılında yaptığı sınıflamada hibrit formlar, LADA (erişkinde yavaş gelişen otoimmün ilişkili diyabet), ketozo meli tip iki diyabet gibi diğer diyabetik formlar da bulunmaktadır.","Monojenik diyabetler, tek gen mutasyonuyla ilişkili diyabet formları, pankreas hastalıklarına bağlı diyabet, endokrin diğer hastalıklara bağlı diyabet ve ilaçlar (özellikle glukokortikoidler) gibi kimyasal nedenler de diyabet formlarını oluşturur.","Gebelikte tespit edilen gestasyonel diyabet de önemli bir diyabet formudur."]},"beginTime":555,"endTime":662,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=555&ask_summarization=1"},{"index":6,"title":"Tip Bir Diyabet","list":{"type":"unordered","items":["Tip bir diyabet genellikle genç yaşlarda tespit edilir ve 6-7 yaş, 12-13 yaş ve 20 yaş civarında üç farklı pik yapar.","Tip bir diyabet tamamen otoimmün aracılığıyla, T-lenfositler aracılığıyla pankreastaki beta hücrelerinin hızlı bir şekilde yok edildiği bir formdur ve tip iki diyabete göre çok daha az görülür.","Tip bir diyabet üç evre olarak ayrılır: preklinik fazda otoimmünite başlar ancak beta hücre hasarı ileri olmadığı için semptom yoktur, ikinci fazda antikor pozitifliği ve prediyabetik belirtiler görülür, üçüncü fazda hastalar semptomatiktir ve standart diyabet tanı kriterleri karşılanır."]},"beginTime":662,"endTime":749,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=662&ask_summarization=1"},{"index":7,"title":"Tip Bir Diyabetin Tanı Kriterleri","list":{"type":"unordered","items":["Tip bir diyabet hastaları akut tanı alır ve bazen ketosidozla gelirler, bu nedenle tanı anında hemoglobin A1C'leri henüz yükselmemiş olabilir.","Tip bir diyabet kuşkusunda hemoglobin A1C yerine açlık plazma glikozunu ölçmek daha yönlendirici olacaktır.","Tip bir diyabettes melts'un tanısında rutin olarak otoantikor ya da C-peptit düzeyinin ölçülmesi önerilmez, ancak emin olunamadığında bu testler yapılabilir."]},"beginTime":749,"endTime":788,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=749&ask_summarization=1"},{"index":8,"title":"C-Peptit ve Tip Bir Diyabet","list":{"type":"unordered","items":["C-peptit, insülin sentezlendiğinde pro-insülinden kopan bir moleküldür ve endojen insülin salgısının bir göstergesi olarak kullanılır.","Tip bir diyabetiklerde tanı andan itibaren C-peptitte ciddi bir düşüklük beklenir."]},"beginTime":788,"endTime":830,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=788&ask_summarization=1"},{"index":9,"title":"Tip İki Diyabet ve LADA","list":{"type":"unordered","items":["Tip iki diyabet genellikle metabolik sendrom eşlik eden kilolu erişkin dönemdeki hastalarda görülür, ancak bazı hastalarda yavaş giden otoimmün bir diyabet (LADA) olabilir.","LADA'dan şüphelenmek için yaş 30'un üzerinde olmalı, genellikle 50 yaşından önce ortaya çıkıyor, adacıkta antikorları pozitif olmalı ve metabolik sendrom komponentlerinin bulunmaması veya hafif düzeyde olması gerekir.","LADA hastalarında diyabet başlangıcında insülin gerektirmiyor, ancak tanıdan sonra 3-6 ay veya 1 yıl sonrasında insülin ihtiyacı mutlaka oluyor."]},"beginTime":830,"endTime":947,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=830&ask_summarization=1"},{"index":10,"title":"LADA Tanısı","list":{"type":"unordered","items":["LADA tanısında öncelikle anti-GAD (anti-glutamik asit dekarboksilaz) taraması yapılır ve genellikle pozitiftir.","Anti-GAD negatifse, anti-insülin antikorları da ölçülebilir.","Serum C-peptit düzeyi düşükse LADA ile ilgili kuşku kalmaz, 2,10 veya üzerindeyse 6 ayda bir kontrol yapılmalıdır."]},"beginTime":947,"endTime":1010,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=947&ask_summarization=1"},{"index":11,"title":"Modi (Erken Başlangıçlı Diyabet)","list":{"type":"unordered","items":["Modi, erken başlangıçlı veya genç başlangıçlı diyabetik hastalarda tek gende bir mutasyon sonucu ortaya çıkan bir durumdur.","Modi hastalarında güçlü aile öyküsü ve mobil uyumlu klinik özellikleri vardır.","Bu hastalarda beta hücre fonksiyonları korunmuş olup, açlık C-peptit düzeyi 0,60'nın üzerinde ve pankreas adacık Otan antikorları negatif olmalıdır."]},"beginTime":1011,"endTime":1093,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1011&ask_summarization=1"},{"index":12,"title":"Modi Tipleri ve Özellikleri","list":{"type":"unordered","items":["Modi'de 13 farklı tip bulunmakta olup, en sık görülen tip 3'tür.","Modi tip 3 hastalarında HNF-1 alfa defekti vardır ve bu hastalarda sülfan üre hassasiyeti çok yüksektir.","Modi tip 3 hastalarının C-reaktif protein düzeyleri diğer modi tiplerine göre daha düşüktür (genellikle 0,75 mg/L'nin altındadır)."]},"beginTime":1093,"endTime":1191,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1093&ask_summarization=1"},{"index":13,"title":"Diyabet Taraması","list":{"type":"unordered","items":["Toplumda diyabet taraması yapılmalıdır; 35 yaşından itibaren üç yılda bir açlık plazma glikozuyla değerlendirme yapılmalıdır.","Beden kitle indeksi 25'ten yüksek olan asemptomatik kişilerde ve risk gruplarında (birinci veya ikinci derece yakınlarında diyabet bulunan kişiler, hipertansif bireyler, dislipidemikler, polikistik over sendromu olan kadınlarda) daha erken tarama yapılabilir.","Prediyabet saptanan bireylerde yılda bir kez, gestasyonel diyabet tanısı almış kadınlarda doğum sonrası diyabet saptanmasa bile üç yılda bir diyabet taraması yapılmalıdır."]},"beginTime":1191,"endTime":1315,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1191&ask_summarization=1"},{"index":14,"title":"Diyabet Hastalarının Bakımı","list":{"type":"unordered","items":["Diyabet hastalarında tanı koyulduktan sonra laboratuvar ve klinik komplikasyonlar değerlendirilmelidir.","Hastalara diyabet eğitimi verilmeli, diyetisyen, ayak bakımı ve diyabet eğitim veren hemşireye yönlendirilmelidir.","Hastalar yıllık izlenmeli, özellikle 3-6 aylık dönemlerde glikohemoglobin kontrolü yapılmalıdır."]},"beginTime":1315,"endTime":1446,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1315&ask_summarization=1"},{"index":15,"title":"Diyabet Komplikasyonları ve İzleme","list":{"type":"unordered","items":["Diyabet komplikasyonları mikrovasküler (nöropati, nefropati, retinopati) ve makrovasküler (koroner arter hastalığı, periferik damar hastalığı, inme) olarak ikiye ayrılır.","Ara kontrollerde kan basıncı, ayak nabız muayenesi, diyetisyen konsültasyonu, hemoglobin A1C, açlık plazma glikoz, SMBG (self-monitoring of blood glucose) ve tam idrar tetkikleri yapılmalıdır.","Serum kreatinin ölçülerek GFR hesaplanmalı ve nefropati komplikasyonu erken tespit edilmelidir."]},"beginTime":1446,"endTime":1564,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1446&ask_summarization=1"},{"index":16,"title":"Diyabet Hastalarında Kan Şekeri Ölçümü","list":{"type":"unordered","items":["Diyabet hastaları mutlaka onaylanmış ve güvenilir kan şekeri ölçüm cihazları kullanmalı, adı sanı duyulmamış cihazlardan uzak durulmalı.","Tip 1 diyabetikler, gebe diyabetikler ve bazal-bolus insülin tedavisi kullanan hastalar günde 3-4 kez öğün öncesinde açlık plazma glikozlarını ölçmelidir.","Bazal-bolus insülin tedavisi alan hastalarda ayrıca haftada bir gece yatarken ve ayda bir sabaha karşı saat 2-4 arasında da ölçüm yapılması önerilir."]},"beginTime":1568,"endTime":1631,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1568&ask_summarization=1"},{"index":17,"title":"Kan Şekeri Ölçümünün Önemi","list":{"type":"unordered","items":["Gece ölçümleri, Smogi (şafak) ve Down (şafak) fenomenlerini ayırt etmek için önemlidir.","Down fenomeninde gece hiperglisemi olur ve sabaha da hiperglisemik çıkılır, sebebi gece artan insülin karşıtı hormonlardır.","Smogi fenomeninde hastalar gece hipoglisemiye girer, daha sonra insülin karşıtı hormonlar artarak sabah saatlerinde reaktif bir hiperglisemi gelişir."]},"beginTime":1631,"endTime":1692,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1631&ask_summarization=1"},{"index":18,"title":"Farklı Diyabet Tedavilerinde Kan Şekeri Ölçümü","list":{"type":"unordered","items":["Sadece bazal insülin tedavisi alan tip 2 diyabetiklerde en az günde bir kez kan şekeri ölçümü yapılması gerekir, ancak farklı saatlerde yapılması önerilir.","Beslenme tedavisi ve oral anti-diyabetik alan tip 2 diyabetiklerde glisemik kontrol düzeyi, tedavi şekli ve kişisel özelliklere göre haftada 3-4 kez kan şekeri ölçümü yapılması önerilir.","Insülin kullanan hastalarda daha yoğun kan şekeri takibi yapılması, tedavinin etkili olup olmadığı ve hipoglisemi riski olup olmadığı için önemlidir."]},"beginTime":1692,"endTime":1751,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1692&ask_summarization=1"},{"index":19,"title":"Tip 1 Diyabetin Önlenmesi","list":{"type":"unordered","items":["Tip 1 diyabet otoimmün bir hastalık olduğu için otoimmüniteyi azaltarak hastalığın gelişimini geciktirmek mümkündür.","Remap (anti-CD20 monoküle antikoru), abatacept ve teblizumab gibi ilaçlar tip 1 diyabetin başlangıcını geciktirdiği gösterilmiştir.","Teblizumab, CD3'ü bloke ederek efektör T hücrelerin aktive olmasını engeller ve bu sayede beta hücrelerine saldırmasını önler."]},"beginTime":1751,"endTime":1902,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1751&ask_summarization=1"},{"index":20,"title":"T Hücrelerinin Aktive Olması Mekanizması","list":{"type":"unordered","items":["T hücresini aktive etmek için T hücre reseptörü (TCR) uyarılması ve CD28'in aktive olması (kostümülasyon) gerekir.","Abatacept, CD80-CD86 inhibitörüdür ve CD28'in aktive olmasının önüne geçerek co-stimülasyonu inhibe eder.","CTLA-4, enerji veya apoptoz reseptörüdür ve T hücresinde aktive edildiğinde hücre apoptoza gider, bu nedenle CTLA-4'ü inhibe ederek T hücrelerinin efektivitesini artırmak kanser tedavisinde kullanılır."]},"beginTime":1902,"endTime":2121,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=1902&ask_summarization=1"},{"index":21,"title":"Tip 2 Diyabetin Fizyopatolojisi","list":{"type":"unordered","items":["Tip 2 diyabet hastalığının fizyopatolojisinde temel olarak insülin direnci vardır.","Süreç içerisinde insülin sekresyon defekti de bu duruma eklenir."]},"beginTime":2121,"endTime":2136,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2121&ask_summarization=1"},{"index":22,"title":"Prediyabetik Hastalarda Tedavi","list":{"type":"unordered","items":["Prediyabetik hastalarda öncelikle yaşam tarzı değişiklikleri ve diyet modifikasyonları önerilmelidir.","Üç-altı ay içerisinde beslenmeye yanıt alınamıyorsa medikal tedavi ile prediyabeti geriletmeye çalışılabilir.","En popüler tedavi metformindir, ayrıca akarboz da endikasyonlu bir ilaçtır."]},"beginTime":2140,"endTime":2195,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2140&ask_summarization=1"},{"index":23,"title":"Metformin ve Akarboz","list":{"type":"unordered","items":["Metformin insülin sensitivitesini arttıran, insülin direncini azaltan ve hepatik glikoz salınımını azaltan bir ilaçtır.","Akarboz alfa glikozidaz inhibitörüdür, yemeklerden önce içilerek karbonhidrat emilimini azaltır.","Metformin kullanımı sırasında B12 eksikliği riski yüksektir ve bu durumda replasman yapılması gerekir."]},"beginTime":2195,"endTime":2253,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2195&ask_summarization=1"},{"index":24,"title":"Glisemik Kontrol Hedefleri","list":{"type":"unordered","items":["Diyabetik hastalarda gliko-hemoglobin A1c'nin %7'nin altına düşürülmesi hedeflenir.","65 yaşın üzerinde, ciddi kardiyovasküler hastalığı olan veya kırılgan hastalarda hipoglisemi riski nedeniyle hedef %8'in altında olabilir.","Genç hastalarda hiperglisemiye bağlı komplikasyon riski düşük olduğu için hedef %6-6,5'in altına çekilebilir."]},"beginTime":2253,"endTime":2441,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2253&ask_summarization=1"},{"index":25,"title":"Metformin'in Özellikleri ve Yan Etkileri","list":{"type":"unordered","items":["Metformin tip 2 diyabetik hastalarda kutsal ilaç olarak kabul edilir ve insülin direncini azaltır.","Metformin'in efektif dozu 2000 mg'dir, ancak başlangıçta 1500 mg veya 2500 mg ile başlanır ve dispeptik şikayetler gelişmezse doz yavaş yavaş artırılır.","Metformin ağızda metalik tat yapabilir, B12 eksikliği ve laktik asidoz (GFR 30'un altındaysa) gibi yan etkileri olabilir."]},"beginTime":2441,"endTime":2615,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2441&ask_summarization=1"},{"index":26,"title":"Metformin'in Kontra Endikasyonları","list":{"type":"unordered","items":["İleri derecede renal fonksiyon bozukluğu, karaciğer ciddi yetersizliği ve laktik asidoz öyküsü olan hastalarda metformin kullanılmamalıdır.","Ciddi hipoksi ortaya çıkartacak hastalıklar (koah), kronik veya akut alkol intoksikasyonu, miyokard enfaktüsü ve ketonomi olan hastalarda metformin kullanılmamalıdır.","Tedavi dirençli konjestif kalp yetmezliği olan hastalarda metformin kullanımı uygun değildir."]},"beginTime":2615,"endTime":2659,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2615&ask_summarization=1"},{"index":27,"title":"Insülin Salgılatıcı İlaçlar","list":{"type":"unordered","items":["Insülin salgılatıcı ilaçlar (insülin sekratogogları) beta hücrelerini uyararak insülin sekresyonu sağlar.","Bu ilaçlar beta hücrelerindeki ATP bağımlı potasyum kanallarını inhibe ederek membran depolarizasyonunu tetikler, voltaj bağımlı kalsiyum kanallarını açar ve hücre içerisinde kalsiyum artışı sağlar.","Türkiye'de en çok kullanılan insülin salgılatıcı ilaçlar arasında sülf üreler (glipizid, glitazon) ve glititler (glipaglitazon) bulunmaktadır."]},"beginTime":2665,"endTime":2741,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2665&ask_summarization=1"},{"index":28,"title":"Insülin Salgılatıcı İlaçların Etkileri ve Yan Etkileri","list":{"type":"unordered","items":["Bu ilaçlar insülin sekrete ettirdikleri için hipoglisemi riski ve kilo alımına sebep olabilirler.","Sülf üreler kliniklere göre daha uzun süre etki gösterir ve günde tek veya iki doz kullanılırken, glititler günde üç kez kullanılmak zorundadır.","Glinitler özellikle postprandiyal (tokluk) glikozunu kontrol altına alma konusunda etkilidir."]},"beginTime":2741,"endTime":2833,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2741&ask_summarization=1"},{"index":29,"title":"Insülin Salgılatıcı İlaçların Kontra Endikasyonları","list":{"type":"unordered","items":["Bu ilaçlar tip bir diyabette, sekonder diyabetik hastalarda, hiperglisemik acil durumlarda ve gebelikte kullanılmamalıdır.","Gebelikte diyabet tanısı koyulduğunda FDA ve EMA endikasyonu olan tek ilaç insülindir, diğer ilaçların endikasyonu yoktur.","Ağır enfeksiyon, sülf üreye karşı alerji, hipoglisemi yatkınlığı, dekompanze karaciğer veya son dönem böbrek yetmezliği olan hastalarda bu ilaçlar kullanılmamalıdır."]},"beginTime":2833,"endTime":2928,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2833&ask_summarization=1"},{"index":30,"title":"Glitazon Grubu İlaçlar","list":{"type":"unordered","items":["Glitazon grubu ilaçlar (pioglitazon, rosiglitazon) insülin duyarlılığını arttırır, insülin direncini azaltır ve hepatik glikoz salınımını azaltır.","Bu ilaçlar metforminden farklı olarak kilo aldırır ve su tutulumunu arttırır.","Rossiglitazon, kardiyovasküler ölüm riskinin daha yüksek olduğu gösterildiği için Türkiye ve Avrupa'da kullanılmamaktadır."]},"beginTime":2928,"endTime":3079,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=2928&ask_summarization=1"},{"index":31,"title":"İlaç Seçimi","list":{"type":"unordered","items":["Diyabetik hastalarda ilaç seçimi yaparken gliko-hemoglobin değerini düşürme kapasitesi önemli bir faktördür.","İlaç seçimi yaparken hastaya zarar verme riski ve ekstra faydaları da göz önünde bulundurulmalıdır.","Örneğin, kilolu veya obez hastalarda sülf üreleri gibi ilaçlar tercih edilebilir."]},"beginTime":3079,"endTime":3161,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3079&ask_summarization=1"},{"index":32,"title":"Oral Antidiyabetiklerin Yan Etkileri ve Kullanım Alanları","list":{"type":"unordered","items":["Sulfe'nin üreler oral antidiyabetikler içerisinde gliko-hemoglobini en fazla düşürenlerden biridir, ancak obez hastalara kilo verdirecek bir ilaç vermek istenmez.","Kalp yetmezliği olan hastalara glitazon verilmemelidir çünkü bu ilaçlar hospitalizasyon riskini artırır, ödemi arttırır ve hasta yüklenme bulgusunu artırır.","Kalp yetmezliği olan diyabetik hastalarda mortaliteyi azaltacak SGLT-2 inhibitörleri veya kilo verdirecek GLP-1 reseptör analogları tercih edilmelidir."]},"beginTime":3162,"endTime":3272,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3162&ask_summarization=1"},{"index":33,"title":"Glitazonların Yan Etkileri ve Kontra Endikasyonları","list":{"type":"unordered","items":["Glitazonlar konjestif kalp yetmezliği, kardiyovasküler olay riskinde artış, greft soft termopatisini alevlendirme ve kırık riskinde artışa yol açabilir.","Post menopozal kadınlarda ve osteoporozu olan hastalarda glitazon grubu ilaçlar kullanılmamalıdır.","Glitazonlar makule ödeminde ödemi daha da arttırarak görme kaybını şiddetlendirebilir ve mesane kanseri riskini artırabilir."]},"beginTime":3272,"endTime":3363,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3272&ask_summarization=1"},{"index":34,"title":"Alfa Glikozi Doz Inhibitörleri","list":{"type":"unordered","items":["Alfa glikozi doz inhibitörleri glikozi dozu inhibe ederek polisakkaritlerin parçalanmasını engeller ve glikoz emilimini azaltır.","Bu ilaçlar şişkinlik, hazımsızlık ve nadiren demir eksikliği anemisi yapabilir.","Alfa glikoz inhibitörleri genellikle gastrointestinall sistemle ilişkili kontra endikasyonlara sahiptir."]},"beginTime":3363,"endTime":3399,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3363&ask_summarization=1"},{"index":35,"title":"GLP-1 Reseptör Agonistleri","list":{"type":"unordered","items":["GLP-1 reseptör agonistleri mükemmel bir ilaç grubudur ve metabolik etkileri vardır; yaşlanmayı yavaşlattıkları gösterilmiştir.","Bu ilaçlar insülin tropik peptit agonisti etkisi de olan dual etkili ilaçlar da çıkmaya başlamıştır.","GLP-1 reseptör agonistleri genellikle supkutan kullanılır, ancak semaglutit oral formu da vardır."]},"beginTime":3399,"endTime":3512,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3399&ask_summarization=1"},{"index":36,"title":"GLP-1 Reseptör Agonistlerinin Yan Etkileri ve Kontra Endikasyonları","list":{"type":"unordered","items":["GLP-1 reseptör agonistleri bulantı, kusma, konstipasyon, karın ağrısı, pankreatit ve safra taşı oluşumu riskinde artış yapabilir.","Bu ilaçlar gastroparazi ve ilius yapabilir, ayrıca C-hücre hiperplasi yaptığı gösterildi ve medüller tiroid kanseri riskini arttırabilir.","Pankreatik pankreas kanseri öyküsü, aşikar ciddi bir gastrointestinall hastalığı, medulla tiroid kanseri öyküsü veya MEE öyküsü olan hastalarda kullanılmamalıdır."]},"beginTime":3512,"endTime":3612,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3512&ask_summarization=1"},{"index":37,"title":"DPP-4 Inhibitörleri","list":{"type":"unordered","items":["DPP-4 inhibitörleri GLP-1'i yıkan enzimdir ve bu enzimi inhibe ettikleri için endojen GLP-1 etkisini arttıran bir ilaç grubudur.","Bu ilaç grubu en fazla kilo verdiren ilaç grubudur, kardiyovasküler olay riskini azaltır ve diyabetik nefropati progresyon riskini azaltır.","DPP-4 inhibitörleri kilo üzerine nötr etkilidir ve lina gliptin böbrek yetmezliğinde doz ayarı gerektirmeyen popüler bir ilaçtır."]},"beginTime":3612,"endTime":3703,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3612&ask_summarization=1"},{"index":38,"title":"DPP-4 Inhibitörlerinin Yan Etkileri","list":{"type":"unordered","items":["DPP-4 inhibitörleri üst solunum yolu enfeksiyonu benzeri yakınmalar yapabilir.","Saksı gliptin kalp yetmezliği olan hastalarda hospitalizasyonun riskini arttırdığı gösterildi, bu nedenle bu hastalarda verilmemelidir.","Alogliptin de kalp yetmezliğinde hospitalizasyon riskini hafifçe arttırdığı gösterildiği için bu hastalarda tercih edilmemektedir."]},"beginTime":3703,"endTime":3711,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3703&ask_summarization=1"},{"index":39,"title":"DPP-4 Inhibitörleri ve Yan Etkileri","list":{"type":"unordered","items":["Saksı gliptin kalp yetmezliği nedeniyle hastaneye yatışı riskini arttırıyor.","DPP-4 inhibitörleri pankreatit riskini arttırabiliyor ve pankreatit öyküsü olan hastalarda kontra endikedir.","Bu ilaçlar ciddi deri hastalıkları (bluz penfigoid, kütaz vaskülit) ortaya çıkabilir ve gebelikte, ciddi karaciğer yetmezliğinde (GFR 15'in altında) ve kalp yetmezliğinde kullanılmamalıdır."]},"beginTime":3715,"endTime":3788,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3715&ask_summarization=1"},{"index":40,"title":"SGLT-2 Inhibitörleri","list":{"type":"unordered","items":["SGLT-2 inhibitörleri (dapagliflozin, empagliflozin) kilo kaybına sebep olabiliyor ve özellikle kilolu hastalarda tercih sebebi olabilir.","Bu ilaçlar hipertansif hastalarda kan basıncını düşürebiliyor, serum ürik asit düzeyini düşürüyor ve albümleri azaltıyor.","SGLT-2 inhibitörleri nefropati progresyonunu azaltıyor ve kalp yetmezliğinde mortaliteyi azalttığı gösterildi."]},"beginTime":3788,"endTime":3957,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3788&ask_summarization=1"},{"index":41,"title":"SGLT-2 Inhibitörlerinin Yan Etkileri","list":{"type":"unordered","items":["SGLT-2 inhibitörleri poliüri (sıvı kaybı) yapıyor, kan basıncını düşürebiliyor ve hipotansiyon riski var.","Bu ilaçlar öglisemik ketosi dozu yapabiliyor, normalde 250'nin üzerinde plazma glikozunda görülen ketosidoz, bu hastalarda 150 civarında görülebiliyor.","En sık görülen yan etkilerinden biri genti ürünler enfeksiyonları, özellikle vajinal kandidiyazis ve nadiren Fournier gangreni yapabiliyor."]},"beginTime":3957,"endTime":4074,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=3957&ask_summarization=1"},{"index":42,"title":"Tip 2 Diyabet Tedavisinde İlaç Seçimi","list":{"type":"unordered","items":["Tip 2 diyabetik hastalarda yaşam tarzı değişiklikleri ve metformin ile başlanmalı, ek ilaç verilecekse kardiyovasküler risk faktörleri varsa GLP-1 reseptör agonistleri veya SGLT-2 inhibitörleri tercih edilmeli.","Obez hastalarda kilo verdirecek bir ilaç vermek isteniyorsa GLP-1 reseptör agonistleri veya SGLT-2 inhibitörleri kullanılabilir.","Düşük hipoglisemi riski isteniyorsa glitazonlar, GLP-1 agonistleri, DPP-4 inhibitörü veya SGLT-2 inhibitörü kullanılabilir."]},"beginTime":4074,"endTime":4143,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4074&ask_summarization=1"},{"index":43,"title":"İlaçların Gliko-Hemoglobin Düşürme Etkisi","list":{"type":"unordered","items":["Sülfenin üreleri, metformin, glinitler, GLP-1 reseptör agonistleri ve SGLT-2 inhibitörleri hemoglobin A1C'yi iyi azaltıyor, DPP-4 inhibitörleri ve alfa-glukozidaz inhibitörleri daha zayıf kalıyor.","Monoterapi olarak kullanıldığında bu ilaçlar yaklaşık %1-2 oranında hemoglobin A1C düşürüyor.","Mevcut tedaviye eklendiğinde bu ilaçların gliko-hemoglobin düşürücü etkisi azalıyor, ikinci ilaç olarak eklendiğinde %0,5-1, üçüncü ilaç olarak eklendiğinde %0,3 civarında düşürüyor."]},"beginTime":4143,"endTime":4281,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4143&ask_summarization=1"},{"index":44,"title":"Insülin Endikasyonları","list":{"type":"unordered","items":["Tip 1 diyabet hastalarında insülin temel tedavidir ve insülinsiz tedavi düşünülemez.","LADA grubunda (otoimmün erişkin başlangıçlı yavaş giden otoimmün diyabet) birkaç ay sonrasında insülin gereklidir.","Tip 2 diyabette melitusu olan hastalarda gliko-hemoglobin %9'un üzerindeyse, ağır hiperglisemik sendrom, diyabetik ketositoz, hiperglisemik hiperrozmalar koması veya akut miyokard enfarktüsü durumunda insülin başlanmalıdır.","Gebelikte oral antidiyabetiklerin endikasyonu olmadığı için temel olarak insülin kullanmak mantıklıdır."]},"beginTime":4286,"endTime":4359,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4286&ask_summarization=1"},{"index":45,"title":"Insülin Türleri","list":{"type":"unordered","items":["Insülinler hızlı etkili, orta etkili ve uzun etkili bazal insülinler olmak üzere üç ana gruba ayrılır.","Hızlı etkili insülinler arasında regüler insülin yerine analog insülinler (insülin glisin, aspart ve lispro) kullanılır.","Bazal etkili insülinler orta uzun ve çok uzun etkili olarak gruplandırılır; orta etkili MPH, uzun etkili detamir ve glar insülini, çok uzun etkili ise deguludec ve icodec insülinleri vardır.","Karışım insülinlerde hızlı etkili insülin ve orta-uzun etkili insülin belli oranlarda karıştırılır, örneğin MPH ve regüler insülin %70-%30 oranında karıştırılabilir."]},"beginTime":4359,"endTime":4515,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4359&ask_summarization=1"},{"index":46,"title":"Tip 1 Diyabet Tedavisinde Insülin Kullanımı","list":{"type":"unordered","items":["Tip 1 diyabet tedavisinde insan fizyolojisini taklit etmek için günde tek veya iki doz uzun etkili insülin (detamir, glasi, degludec) bazal sekresyon taklit eder.","Glikozla simüle edilmiş insülin salgısını taklit etmek için her öğünle beraber hastaya hızlı etkili insülin analoğu (aspart, gulilisin, lispro) günde üç kez verilir.","Bu tedavi şemasına bazal bos insülin şeması veya intensif insülin tedavisi denir."]},"beginTime":4515,"endTime":4622,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4515&ask_summarization=1"},{"index":47,"title":"Insülin Başlangıç Dozları","list":{"type":"unordered","items":["Yeni başlayan tip 1 diyabetik hastalarda insülin başlangıç dozu kiloya 3,4 civarından hesaplanır ve bu değer ikiye bölünür.","Hesaplanan insülin dozunun yarısı bazal olarak verilir, geriye kalan yarısı öğünlere bölünür.","Hastalara kalori sayma ve karbonhidrat sayma eğitimi verilir, bu sayede hastalar insülin dozlarını öğünündeki kaloriye ve spora göre ayarlayabilir.","Normal kilolu hastalarda fiziksel aktivitesine göre insülin dozu hesaplanır, aktivite fazlaysa doz düşürülür, azaldıkça doz artırılır.","Obezite hastalarında fiziksel aktivite yoğunluğuna göre insülin dozu 0,5 ile 8 arasında değişir.","Böbrek yetmezliği olan hastalarda insülin dozu azaltılmalıdır çünkü insülinin etki süresi uzar."]},"beginTime":4622,"endTime":4772,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4622&ask_summarization=1"},{"index":48,"title":"Insülin Pompa Tedavisi","list":{"type":"unordered","items":["Bazal borus insülin tedavisi yerine daha iyi bir tedavi olan insülin pompa tedavisi vardır.","Insülin pompa tedavisi subkutan bir cihazla uygulanır ve fizyolojiyi taklit ederek insülin verir, hipoglisemi riskini dramatik olarak azaltır.","Sensör takılan pompalar kan glikozunu ölçerek insülin dozunu ayarlayabilir ve hipoglisemi riskini çok azaltır.","Insülin pompa tedavisinde hastanın entelektüel düzeyi iyi olmalı, cihazı kullanabilecek ve teknoloji ile barışık olmalıdır."]},"beginTime":4772,"endTime":4862,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4772&ask_summarization=1"},{"index":49,"title":"Tip 1 Diyabet Tedavisi","list":{"type":"unordered","items":["Tip 1 diyabet hastalarında analog insülinler (lispro, aspart, glulisin) kristalize insüline göre daha az hipoglisemiye sebep olduğu için tercih edilmektedir.","Sensör destekli insülin pompası, deneyimli merkezler tarafından takip edilmesi koşuluyla seçilmiş hastalarda ve nokturnal hipoglisemileri olan hastalarda iyi bir tedavi yöntemidir."]},"beginTime":4866,"endTime":4915,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4866&ask_summarization=1"},{"index":50,"title":"Antidiyabetik İlaçların Etkileri","list":{"type":"unordered","items":["Sülfenin üreler, oral antidiyabetikler arasında glikohemoglobini en fazla düşüren ilaçlardır ancak hipoglisemi riski daha fazladır.","Sülfenin üreler, glitazonlar ve insülin vücut ağırlığı üzerine olumsuz etki yaparken, GLP-1 reseptör agonistleri ve SGLT-2 inhibitörleri kilo verme etkisi gösterir.","GLP-1 reseptör agonistleri kardiyovasküler olay riskini azaltıcı etkisi dramatiktir, SGLT-2 inhibitörleri ise özellikle kalp yetmezliği ile ilgili koruyucu etkileri vardır."]},"beginTime":4915,"endTime":5004,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=4915&ask_summarization=1"},{"index":51,"title":"Kronik Böbrek Hastalığı ve Antidiyabetik İlaçlar","list":{"type":"unordered","items":["Diyabetik nefropatisi olan hastalarda progresyonu azaltan iki önemli ilaç grubu vardır: ilk basamakta SGLT-2 inhibitörü, progresyon devam ediyorsa GLP-1 reseptör agonisti.","İleri yaşta olan insanlarda DPP-4 inhibitörleri güvenli ve yan etki profili masumdur.","Kardiyovasküler hastalığı olanlarda SGLT-2 inhibitörleri ve GLP-1 reseptör agonistleri tercih edilmelidir."]},"beginTime":5004,"endTime":5072,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5004&ask_summarization=1"},{"index":52,"title":"Özel Durumlarda İlaç Seçimi","list":{"type":"unordered","items":["Hiperlipidemi (dislipidemi) durumunda en faydalı olan metformindir.","Metabolik disfonksiyon ilişkili yağlı karaciğer hastalığı (eskiden nanokolik yağlı karaciğer hastalığı olarak bilinir) durumunda GLP-1 reseptör agonistleri ve glitazonlar başarılıdır.","Kronik böbrek yetmezliği olan hastalarda GFR 30'un altındaysa SGLT-2 inhibitörleri kullanılabilir, ancak GFR 45'in altındaysa glikoz düşürücü etkisi azalır."]},"beginTime":5072,"endTime":5155,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5072&ask_summarization=1"},{"index":53,"title":"Yeni Tanı Tip 2 Diyabet Tedavi Algoritması","list":{"type":"unordered","items":["Glikohemoglobin 8'in altındaysa monoterapi ile başlanabilir, kontrendikasyon yoksa metformin tercih edilir.","Glikohemoglobin 8-9 arasındaysa metforminle kombine başka bir antidiyabetik eklenir, bazen üçlü tedavi de uygulanabilir.","Glikohemoglobin 9'un üstünde ise insülin başlamak gerekir, genellikle metformin artı insülin şeklinde başlanır."]},"beginTime":5155,"endTime":5264,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5155&ask_summarization=1"},{"index":54,"title":"Tedavi Takibi ve Modifikasyon","list":{"type":"unordered","items":["Tedavilerden üç ay sonrasında mutlaka glikohemoglobin kontrolü yapılmalıdır.","Monoterapi almasına rağmen glikohemoglobin 8'in altına indirilemiyorsa, ikinci veya üçüncü bir oral antidiyabetik veya insülin eklenebilir.","Glikohemoglobin 8-9 arasındaysa ve ikili veya üçlü tedavi kullanılıyorsa genellikle insülin eklenmesi tercih edilir."]},"beginTime":5264,"endTime":5326,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5264&ask_summarization=1"},{"index":55,"title":"Özel Durumlarda İlaç Seçimi","list":{"type":"unordered","items":["Aterosklerotik kalp hastalığı varsa metformin ilk basamakta verilir, ek olarak GLP-1 reseptör agonisti veya SGLT-2 inhibitörü kullanılabilir.","Kalp yetmezliği varsa özellikle SGLT-2 inhibitörleri tercih edilir, GLP-1 reseptör agonisti de eklenebilir.","Kronik böbrek hasarı varsa özellikle SGLT-2 inhibitörlerinin koruyucu etkisi çok fazladır, ilk basamakta metformine ek olarak SGLT-2 inhibitörü eklenir."]},"beginTime":5326,"endTime":5380,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5326&ask_summarization=1"},{"index":56,"title":"Obez Hastalarda Diyabet Tedavisi","list":{"type":"unordered","items":["Obez hastalarda kilo verdirme etkisi yoğun olan ilaç grubu GLP-1 reseptör agonistleri olup, en fazla kilo verdirenler semaglutit ve tirzepati'dir.","Tirzepati hem GLP-1 reseptör agonisti hem de glikoz bağımlı insülinotropik peptiti uyaran agonistik etkisi olan dual etkili bir ilaçtır.","Kilo etkisi olumsuz olan dört temel ilaç grubu: insülinler, glitazonlar, sulfeni üreler ve glinitlerdir."]},"beginTime":5382,"endTime":5510,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5382&ask_summarization=1"},{"index":57,"title":"Bariatrik Cerrahi","list":{"type":"unordered","items":["Bariatrik cerrahi, tip 2 diyabetli hastalarda çok faydalı bir tedavi yöntemidir ve hastaların diyabeti kontrol altına girmesine yardımcı olur.","Bazı klinik araştırmalarda hastaların yarısında diyabet tamamen ortadan kalkabilmektedir.","Bariatrik cerrahi endikasyonu: vücut kitle indeksi 40 veya üzerindeyse direkt endikasyon, 35 üzerindeyse yeterli medikal tedaviye rağmen optimal glisemik kontrol sağlanamadığında cerrahi endikasyon olur."]},"beginTime":5510,"endTime":5609,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5510&ask_summarization=1"},{"index":58,"title":"Diyabetik Ketoasidoz","list":{"type":"unordered","items":["Diyabetik ketoasidoz, özellikle tip 1 diyabetik hastalarda ilk prezentasyon olarak görülebilir ve hastaların yaklaşık %20-25'i ilk tanı anında ketosidozla gelir.","Ketoasidozun tipik semptomları: halsizlik, bulantı, kusma, ağız kuruluğu, karın ağrısı, nefes darlığı, kilo kaybı ve bilinç bulanıklığıdır.","Hastaların fizik muayenesinde taşikardi, dehidratasyon bulguları, hipotansif olma ve kusma solunumu görülebilir."]},"beginTime":5609,"endTime":5717,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5609&ask_summarization=1"},{"index":59,"title":"Ketoasidoz Tanısı ve Tedavisi","list":{"type":"unordered","items":["Ketoasidoz tanısında parmak ucu glikozunun 250'nin üzerinde olması beklenir, ancak SGLT-2 inhibitörü kullanan hastalarda glikoz normal değerlere yakın olsa bile ketosidoz tablosu ortaya çıkabilir.","Tanıda arterial pH'nın azalması, serum karbonatın azalması, idrar ketonunun pozitif olması ve serum ozmolaritesinin 320'nin üzerinde olması önemlidir.","Ketoasidozda potasyum değeri en çok dikkat edilmesi gereken elektrolittir çünkü insülin yetmezliğinde potasyum düşük olabilir ve insülin tedavisi altında daha da düşebilir."]},"beginTime":5717,"endTime":5863,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5717&ask_summarization=1"},{"index":60,"title":"Ketoasidozun Nedenleri ve Tedavisi","list":{"type":"unordered","items":["Ketoasidozu tetikleyen faktörler arasında enfeksiyonlar, insülin yetmezliği, akut ketobolik durumlar ve akut travmatik durumlar bulunmaktadır.","SGLT-2 kullanan hastalarda nedeni belirsiz bir şekilde ketosidoz tablosu ortaya çıkabilir ve plazma glikoz değeri normale yakın olabilir.","Ketoasidoz tedavisinde ilk basamakta ciddi sıvı defekti olduğu için hızlı bir şekilde sıvı tedavisi başlanmalı, ikinci sırada insülin tedavisine geçilmelidir."]},"beginTime":5863,"endTime":5967,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5863&ask_summarization=1"},{"index":61,"title":"Potasyum Kontrolü ve Insülin Tedavisi","list":{"type":"unordered","items":["Hastanın potasyumu düşükse insülin tedavisi başlatıldığında potasyum daha da düşebilir ve hipopotasemi belirtileri (kardiyak aritmi) ortaya çıkabilir.","Hastanın potasyumu 3,30'un altındaysa öncelikle potasyum düzeltilmeli ve 3,30'un üzerine çıkmadan insülin başlanamaz.","Normal potasyum sınırlarında bile insülin ile beraber potasyum replasmanı verilmelidir çünkü insülin başladığında potasyum düşmeye başlar."]},"beginTime":5972,"endTime":6034,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=5972&ask_summarization=1"},{"index":62,"title":"Insülin Tedavisi Yöntemi","list":{"type":"unordered","items":["Insülin tedavisinde öncelikle bazal insülin (bos insülin) verilir, ardından kiloya 0,10'dan bonus verilir (örneğin 70 kiloya 7 ünite bonus).","Saatlik infüzyon açılır ve ilk saatte plazma glikozu %10'dan fazla düşmüyorsa insülin dozu yetersizdir ve arttırılmalıdır.","Glisemi 200-250'nin altına düştüğünde ve ketosidoz devam ediyorsa tedaviye dekstroz eklenmelidir, aksi takdirde insülin infüzyonu kesilemez ve hipoglisemiye yol açabilir."]},"beginTime":6034,"endTime":6113,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6034&ask_summarization=1"},{"index":63,"title":"Ketosidoz Tedavisi ve Hipoglisemi","list":{"type":"unordered","items":["Ketosidoz düzelince (karbonat yükseliyor, pH 7,30'un üzerine çıkıyor, idrar ketonları negatifleşiyor) oral beslenme açılabilir ve cilt altı insülin verilir.","Cilt altı insülin verildikten sonra infüzyona 2 saat daha devam edilip kesilir, sürekli infüzyonla gitmez.","Bir karbonat tedavisinde çok seçici olunmalı, sadece pH 7'nin altındaysa verilmelidir çünkü yüksek dozlarda olumsuz metabolik etkileri olabilir."]},"beginTime":6113,"endTime":6199,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6113&ask_summarization=1"},{"index":64,"title":"Hipoglisemi ve Tedavisi","list":{"type":"unordered","items":["Hipoglisemi, diyabetik hastalarda kan şekerinin 70'in altına düşmesidir ve normal insanlarda 70'in altına düştüğünde semptomlar az olabilirken diyabet hastalarında daha fazla görülür.","Hipoglisemiye adrenerjik bulgular (titreme, bulantı, taşikardisi, acıkma, soğuk ter) ilk basamakta ortaya çıkar, daha sonra nöro-glikopik semptomlar (baş ağrısı, konsantrasyon azalması, konfüzyon, koma) görülür.","Erken dönemde bilinci açık ve oral alabilen hastalarda 15-20 gram glikoz (3-4 glikoz tableti, 4-5 kesme şeker, 200 ml meyve suyu) verilmelidir, kompleks karbonhidratlar veya yağ içeriği olan yiyecekler kullanılmamalıdır."]},"beginTime":6199,"endTime":6316,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6199&ask_summarization=1"},{"index":65,"title":"Ciddi Hipoglisemi Tedavisi","list":{"type":"unordered","items":["Bilinci bulanık ve oral olarak alamayacak derecede hasta için paranteral tedavi uygulanır: 100-150 ml %20'lik dekstroz veya 100-250 ml %10'luk dekstroz verilebilir.","Glukagon enjeksiyonu (1 mg) hastanın hayatını kurtaracak en önemli tedavidir ancak geçici bir etkidir ve dekstroz tedavisi de başlamalıdır.","Sulfenin üre grubu ilaçlara bağlı hipoglisemi geliştiyse glukagon vermek yeterli olmayabilir, diaz oksit veya okreotit gibi insülin sekresyonunu azaltan ilaçlar eklenmelidir."]},"beginTime":6316,"endTime":6396,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6316&ask_summarization=1"},{"index":66,"title":"Kronik Komplikasyonlar","list":{"type":"unordered","items":["Koroner arter hastalığı diyabetik hastalarda en önemli mortilite sebebidir ve normal popülasyona göre neredeyse dört kat daha fazla koroner arter olay riski vardır.","Diyabet tek başına koroner arter hastalığı için majör bir risk faktörüdür ve diyabetli tüm hastalar kardiyovasküler risk faktörleri açısından yılda en az bir kez değerlendirilmelidir.","Skor 2 diyabet risk skorlaması sistemi kullanılarak hastanın riski hesaplanabilir ve EKG, ekokardiyografi veya stres testleri gibi tarama testleri yapılabilir."]},"beginTime":6396,"endTime":6505,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6396&ask_summarization=1"},{"index":67,"title":"Mikrovasküler Komplikasyonlar","list":{"type":"unordered","items":["Mikrovasküler komplikasyonlar retinopati, nefropati ve nöropati olarak adlandırılır ve bunların gelişiminde en önemli risk faktörü hiperglisemidir.","Bu komplikasyonları önlemek veya progresyonlarını yavaşlatmak için hastanın hiperglisemisinin iyi kontrol altında tutulması gerekir."]},"beginTime":6505,"endTime":6531,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6505&ask_summarization=1"},{"index":68,"title":"Diyabetik Retinopati","list":{"type":"unordered","items":["Gelişmiş ülkelerde 20-74 yaş aralığında en sık körlük sebebi diyabetik retinopatidir.","Hiperglisemi, hipertansiyon ve dislipidemi mikrovasküler komplikasyonlar açısından ek risk faktörleridir.","Gebelerde retinopati riski artar ve diğer mikrovasküler tutulumlar varsa retinopatinin gelişme riski de artar."]},"beginTime":6532,"endTime":6563,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6532&ask_summarization=1"},{"index":69,"title":"Retinopati Taraması ve Tedavisi","list":{"type":"unordered","items":["Tip 1 diyabetik hastalarda tanıdan beş yıl sonra, tip 2 diyabetik hastalarda ise tanı anında retinopati taramasına başlanmalıdır.","GLP-1 reseptör agonistleri aktif veya orta ciddi retinopatisi olan hastalarda kötüleştirebilir, bu nedenle dikkatli takip edilmelidir.","Retinopatiden korunmak için glisemik kontrol, kan basıncı kontrolü, statin ve fenofibrat kullanımı, düzenli egzersiz yapılması gerekir."]},"beginTime":6563,"endTime":6692,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6563&ask_summarization=1"},{"index":70,"title":"Proliferatif Retinopati Tedavisi","list":{"type":"unordered","items":["Proliferatif retinopati tedavisinde lazer, fotoko-koagülasyon ve anti-VGF tedavisi kullanılır.","Anti-VGF tedavisinde bevosizzumap, rani buzumap ve afli-bercep tedavisi popülerdir.","Proliferatif retinopati hastalarında valsalva manevrası yaptığı egzersizler vitröz hemoroloji riskini artırdığı için yapılmamalıdır."]},"beginTime":6692,"endTime":6728,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6692&ask_summarization=1"},{"index":71,"title":"Diyabetik Nefropati","list":{"type":"unordered","items":["Diyabetik nefropati erken dönemde GFR artışı, bazal membran kalınlaşması ve glomerüler disfonksiyonu ile başlar.","Mikro albümüri fazında süreç geriye döndürülebilirken, makro albümüri fazında geri dönüş olmaz ancak nefropati progresyonu yavaşlatılabilir.","Protein-kreatinin oranı 30'un altındaysa normal, 30-300 arasında mikro albümüri, 300'ün üstünde makro albümüri olarak değerlendirilir."]},"beginTime":6728,"endTime":6822,"href":"/video/preview/4683342242185217156?parent-reqid=1769864725082281-15758656006769250745-balancer-l7leveler-kubr-yp-klg-100-BAL&text=kbddweb&t=6728&ask_summarization=1"},{"index":72,"title":"Nefropati Taraması ve Tedavisi","list":{"type":"unordered","items":["Tip 1 diyabette beş yıl geçtikten sonra, tip 2 diyabette tanı anında nefropati taraması yapılmalıdır.","GFR düşük hastalarda 7 gün içerisinde tekrar ölçüm yapılması gerekir, GFR hızlı azalmaya devam ediyorsa akut böbrek hasarı vardır.","Kronik böbrek hastalığı için üç ay sonra tekrar ölçüm yapılır ve diyabete bağlı olup olmadığı 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